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Acne and its
residues
How to treat acne scars is of
great concern to patients with moderate to severe acne lesions.
Failed or inadequate acne treatment is
major cause in development of scarring. The incidence of scarring is
not known by dermatologists. It is frequently stated that acne scarring
results from severe inflammatory nodulocystis acne lesions
occurring deep in the dermis. Although it may occur with only minor
more superficial acne. It is
very likely that some patients are more prone to facial scars. Acne should
be treated early and adequately. Acne-caused
scar can be natural consequence of inflammatory lesions or result of
self manipulation. Wound healing occurs when healing
mechanisms get involved to correct the injured tissue. Their work
may not be as adequate as it seems and it could not leave the skin
in its original state. Healing speed is especially correlated to blood
supply of the region. The
outcome appears to be scarring of different types and shapes
associated with either hyperpigmentation or hypopigmentation.
Types, prognosis and
pathology
The following wound healing
scenario may explain the pathology. The initial stage is defined by
a structured series involving inflammatory cells. This succession of
stages is arranged by neutrophils. Subsequently, macrophages
elaborate a variety of cytokines, which create an environment prone
to granulation tissue constitution. At last, migration of
fibroblasts occurs followed by proliferation and recapitulation by
depositing new collagen. Simultaneously, formation of new
capillaries is triggered by some factors released in the wound area.
A problem arises when this organized process is disrupted by its
anabolic and catabolic phases. An overreacting wound healing
response may occur, creating a raised scar of fibrotic tissue.
Alternatively, deleted collagen is deficiently replaced and forms a
pitted appearance resembling the surface of a golf ball. In either
case, the scar often is a legacy of skewed healing process.
Pathology of acne spots,
however, have less correlation with anabolic and catabloic
processes, rahter, skin pigmenation changes occur prominantly.
Scarring could be presented
in two types. One is associated with increased tissue formation and
the other is associated with some tissue loss
Scarring with
increased skin tissue:
Excessive scar tissue is
classified either as keloid scars or
hypertrophic lesions. When an imbalance occurs between the anabolic
and catabolic phases of the skin's wound healing process, more
collagen is produced than is degraded, and the scar tissue grows in
all directions, which results in lesions with skin thickness build
up.
Keloid scars:
Keloids are raised, reddish-purple, nodular scars which,
upon palpation, are firmer than hypertrophic scars. Upper
body or angle of the jaw have more tendency to this type facial
scar. Skin of dark-complexions (black skin) is more likely to
develop keloid scars. They
could be seen as thick, raised,lobulated fibrotic plaques. They are
often red or darker (excess skin color) than the surrounding skin.
Once body's healing movements continue to make collagen even after a
wound has healed cause the occurrence of keloid scars. The
periphery of keloids is more densely populated with fibroblasts than
normal skin.
Hypertrophic scars: Very similar to keloids, they might be mistaken, however,
hypertrophic scars remain
within the confines of the original integument injury and may
regress with time. Spontaneous restoration of hypertrophic scars is a
likely phenomenon. Acne scars hardly
present as these type of scarring. The scar tissue is formed as the
result of overzealous collagen formation coupled with limited
collagen lysis during the remodeling phase of wound healing.
Scarring with loss
of tissue:
Acne spots: Flat discolorations present as either red or brown
marks, increase in color and hyperpigmentation is seen. Acne spots
are more likely to fade away either gradually without treatment or
using fading creams.
Ice-pick scarring:
These may be superficial or deep, fairly linear but irregular and
commonly occur on the cheeks, loss of tissue is obviously visible.
They have a pitted appearance resembling the surface of a golf ball.
The marks are more resistant to various treatments scars and the
deeper they are the less chance of their treatment and
the longer it takes to be healed entirely.
Depressed fibrotic scars: They present as large with sharp margins and steep sides. Their base
is firm, white and can not be stretched. Fibrotic scars may
result from severe forms of inflammatory acne such as
cystic lesions.
Atrophic scars macules: Atrophic macules present as soft, distensible,
ivory-white in color and small in size (few millimeter in diameter),
different from brown marks especially in their color. Facial break
outs may produce acne scars that
are thickened or more recurrently depressed.
Facial scars after
cystic or inflammatory acne lesions
can manifest as atrophic, saucerized, boxcar or ice pick scarring.
Prompt attention to these affected areas could be the best mode of
facial scar treatment.
Scarring is the result of improper deposition of collagen and
elastin and insufficient work of wound healing aparatus. Lining
epitheliumis not flat and atrophic but hyperplastic. Healing them
involves stimulation of the healing progression and rebuilding
elastin and collagen tissue. There are controversies over whether
vitamin C serums in their highest tolerable concentration enriched
with potent antioxidants could be helpful in collagen rearrangements
and treatment of
stretch marks and other lesions assoiciated with dermal layer
involvement such as in acne scars.
Prevention:
The more acne inflammation persists the more likely formation of scarring. Serious
inflammatory acne is more
associated with acne scar
development. Treatment early in its course is the best method for acne scars prevention and its development. Dermatologists suggest that acne even in
its severe form can often be alleviated. Control and prevention of
facial blemishes is also feasible by correcting hormonal balance
through a change in lifestyle. These methods mostly rely on
nutrition, exercise, grooming and hygiene styles. Read also
frequently asked questions.
Treatments of body scars,
various scars removal
procedures for facial scar reduction
Most have heard of peels
for treatment of scars,
dermabrasion for acne scars removal, silicone injections, collagen implants and laser
resurfacing because these have been widely publicized in the lay
press. For the patient whose facial acne has
recently cleared, it is helpful to know that facial scars tends
to become less apparent in time and that any consideration of
cosmetic surgery for scar reduction should be deferred for at least
1 year. It should be pointed out that treatment of
body lesions takes longer period of time and the response is not as
early as the ones received when treating face scars, mainly
due to richer microcirculation of skin of the face. Also skin
damages on body are generally less amenable to cosmetic surgery.
Basic mechanism of most different treatments is cell renewal at
epidermal level, even though exact underlying mechanism could be
different. When dealing with various treatments one should be
realistic with reasonable expectations. No scar could be removed
completely, however, you can make its appearance very improved.
Extent, location, direction, depth and type of your body scars all are
determining factors in improvement. Claims for rearranging collagen
fibers in dermis should be looked at with skepticism.
Collagen injectables
or body fat transfer:
Collagen or other soft fiber fillers is
injected under the skin to fill out certain types of superficial and
soft deep scars. Collagen treatments of facial scars usually
does not work as well for ice-pick scarring and keloids. Bovine
collagen cannot be used in people with autoimmune diseases. Fat
transfer is helpful for those allergic to bovine (cow-derived)
collagen. The result usually lasts 3 to 6 months. Further collagen
treatments to maintain the cosmetic benefit are done at additional
cost. Fat injection may last somewhat longer. Treatment areas may remain lumpy for months as a result of uneven distribution
following fat injection. Revision of facial scar with collagen injectables or other
soft fiber fillers may be associated with allergy or incorrect
positioning. Superficial and atrophic acne scars as well
as soft scars may do
well. Keloids and ice-pick scars tissue
do not benefit. Collagen treatments of scars or fat
injectables results are temporary and the injections must be
repeated every six months.
Laser scar
resurfacing treatment. Laser has lately opened its doors on treatment of acne lesions. However, this application of laser is still too new, its
prospects seem to be more promising compare to some other type of
treatments. Whether severe types of acne could be
cured by laser remains questionable. Laser for removal of acne lesions
related skin damages appear to be more successful in convex areas of
the face. Lateral cheek and the temples do not benefit from this
technique as much. Laser resurfacing could be associated with
hyperpigmentation (skin color increase) or hypopigmentation for
short or long term in treatment areas. Scarring, cutaneous macules, infection and persistent redness
perioral hypertrophic scars are
also possible side effects of most procedures/treatments with treatment of
laser for facial scars. Some
studies suggest that high-energy, pulsed CO2 laser can safely and
sufficiently improve or even eliminate atrophic scars and
provides many benefits over traditional treatment methods for resurfacing of the skin. That laser resurfaces acne scars is an
idea in need of more discussion. Please visit our page for skin
resurfacing. Which treatments can achieve more effective
results?
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